The answer is “Yes, it is.” It has been confirmed that gout is a form of arthritis. Moreover, it is one of the most severe forms of arthritis. For more information about types of arthritis, read our article.
- 1) The definition of gout
- 2) What led to gouty arthritis?
- 3) General signs and symptoms of gouty arthritis
- 4) Diagnostic Methods
- 5) Treatments
The definition of gout
When the levels of uric acid in the blood are higher than normal, it may result in gout. Gout seems to suddenly, usually in the form of a big toe joint pain or swelling of the lower body. In fact, that is the end result of a process that has been taking place in the body for some time.
Gout often happens with your big toe first. An attack of gout can occur suddenly, often wakes you up in the middle of the night with the feeling that your big toe is on fire. The affected joint becomes too hot, swollen, tender and painful to endure even the light sheet.
- The process of developing gout
Gout usually develops after several years of accumulation of uric acid crystals in the joints and surrounding tissues. A gout attack usually begins with pain in the night has grown worse. A gout attack usually causes pain, swelling, redness, and warmth (inflammation) in the joints, often the big toe. Later, the symptoms gradually disappeared. Most gout attacks stopped after about a week.
- Mild attacks can be stopped after a few hours or 1-2 days. These attacks are often misdiagnosed as tendinitis or sprained.
- Severe attacks can last up to several weeks, the pain lasts for up to 1 month.
- Many people have a second attack of gout within 6 months to 2 years after their first attack. But there may be a period of years between attacks. If untreated gout is, the frequency of attacks often increases over time.
There are several stages of gout:
- 1st stage – hyperuricemia is asymptomatic period before the first gout attack. Although you cannot see any different symptoms, inside, the uric acid levels may increase gradually.
- 2nd stage – Acute gout, or an attack of gout, which occurs when something (such as a night of drinking) causes spike uric acid levels or jostles the crystals were formed in an joints, triggering the attacks. Uric acid crystals begin to form. You start with gout. After an attack, the affected joint to normal sensation. The time between the attacks could grow shorter. The inflammation and pain often strikes at night and enhance the next eight to 12 hours. These symptoms subside after a few days and could disappear within a week to 10 days. Overall, 84% can have an attack within three years. Attacks your future may be more severe, last longer, and involve more than one part.
- 3rd stage – Interval gout is the time between the attacks. Although there is no pain, gout is not going. Low levels can cause harm arthritis. They can affect many companies.
- 4th stage – Chronic gout: In this stage, the attacks became more frequent and the pain may not go away as it used to. Joint damage can occur, which can lead to a loss of mobility. However, this stage can be prevented by appropriate treatments.
- Objects affected
Gout affects more than two million Americans – most men are usually between the ages of 40 and 50, who are overweight, people who regularly drink alcohol, and use diuretics to lower blood pressure and deal with heart problems. In addition to diuretics, there are some drugs that cause the risk of developing gout by flushing out uric acid to reduce the body’s ability.
What led to gouty arthritis?
Gout results from deposits of abnormal uric acid crystals in the joint cartilage. The crystals are then released into the synovial fluid.
Our bodies all have a breakdown product called urate (or uric acid). Most urate (about three quarters) are produced by the body but part comes from the diet of us. Urate stems from a breakdown of substances called purines and often come out mainly in our urine (75%) and to a lesser extent through the small intestine. If urate not easily pass out of the body, or if you produce too much, the level can go on an important point of focus is called “saturation point”. If urate levels are still above the important point is enough of it to precipitate the sodium urate crystals. These special cool around the joints formed at the ends of our arms and legs – feet, knees, fingers, wrists and elbows. Once you have enough crystals in a letter some may spill over from the cartilage where they form and trigger a painful episode of arthritis. In addition, the crystals in the cartilage and bone formation can together to form a solid mass ( ‘tophi’) which gradually increases in size. This causes irreversible joint damage and pain after a day when the joints are used. Purines are found naturally in the body and in some foods such as shellfish, red meat and offal and beverages such as beer and stout. Regularly eating more food and drink purinerich can increase urate concentrations. High fructose levels in many fizzy drinks also increase urate concentrations. Chronic kidney disease is an important cause of increased urate levels but many drugs reduce renal excretion of urate include diuretics and other drugs used to control blood pressure.
It does not mean that everyone with high urate levels will certainly develop gout. However, some people either uric acid overproduction or they produce a normal number, but their kidneys are unable to process it effectively, and an excess of uric acid buildup. We do not know why some people are especially prone to form crystals although osteoarthritis joints forming crystals easier normal joints.
The exact cause of hyperuricemia sometimes unknown, although genetic factors (gene) seems to play a role. Uric acid can form crystals accumulate in the joints. This causes pain and other symptoms.
- Increase Risk factors
- Gene: If family members have gout, you are more likely to develop it.
- Drugs: diuretics or “water pills” given to high blood pressure can increase the concentration of uric acid; so can some drugs inhibit immune system rheumatoid arthritis and psoriasis patients
- Gender and age: Gout occurs more often in men (between the age of 30 and 50). However, after menopause, uric acid levels of women approaching men.
- Diet: Eating a diet that is high in meat and seafood and drinks high in sugary fruit (fructose) encouraging higher levels of uric acid, which increases the risk of gout.
- Obesity: If you are overweight, your body produces uric acid and your kidneys have a more difficult time removing uric acid, significantly increase the risk of gout.
- Medical or health triggers of gout: include untreated high blood pressure and chronic diseases such as diabetes, metabolic syndrome, heart disease and kidney injury part; infection; Take the drug cyclosporine; Starting treatment reduced uric acid; Valence or or in bypass of surgery
- Lifestyle causes gout:
- Crash diets and fasting
- Drinking too much alcohol: For most people, more than two glasses of wine or two bottles of beer a day can increase the risk of gout.
- Eat mostly foods high in purines certain (red meat or shellfish): A big meal fatty acids or an excessive amount of beer and spirits. This may enhance the reaction between the crystal and the lining of your business to trigger the onset of inflammation
- Dehydration (not getting enough fluids)
- Sugary soda: The fructose in fresh soda has recently been shown to increase the risk of gout.
- Your body is stressed by severe illness (eg, pneumonia), fever or surgery. Or if you hurt a portion containing the crystal that it can “shake loose” crystals and trigger an attack.
- Inflammation of calcium pyrophosphate crystal arthritis ( “pseudogout”) is a condition similar to gout, where the surprise attack of inflammation affecting the part.
General signs and symptoms of gouty arthritis
Occur suddenly without any warning and often at night, signs and symptoms of gout include:
- Intense joint pain. Gout usually affects the large joint of the big toe as the first symptom. The pain may be most severe within the first four to 12 hours after it started. It can deteriorate quickly, last for hours, and will be strong that even light pressure from a sheet is unacceptable.
- Lingering discomfort. After the most severe pain subsided, some joint discomfort can last from a few days to a few weeks. Then the attacks will probably last longer and affect more joints. Gout usually affects one part at a time, but if not treated can affect many joints. Joint pain is used to resolve in a week to 10 days can be a lighter, but constant pain.
- Inflammation and redness. The affected joints become swollen, tender, warm and red.
- Limited range of motion. The mobility of joint reduces
- Recurrent gout.
- Enhance gout. Untreated gout can cause deposits of urate crystals in the form of subcutaneous nodules called tophi (TOE-fie), which is not painful, but become swollen and painful gout attacks.
- Kidney stones.
- Call doctor for help:
- It is essential to visit your doctor if the sudden and severe pain in joints comes to you or you have a fever, it may be the result of infection.
- Swollen joints with warm tender, red skin over them.
The important thing is you have to see a doctor even if gout pain has stopped. The accumulation of uric acid cause gout attacks of you may still be stimulating your joints and can eventually cause serious damage.
Gout cannot simply be diagnosed from a blood test, because many people have elevated uric acid levels, but no gouty arthritis. However, doctors are also some tests to diagnose gout may include:
- Check the liquid part. Your doctor may use a needle to draw fluid from your affected joint. When observed under a microscope, your joint fluid may reveal urate crystals. The fluid is observed under a microscope for uric acid crystals. The liquid is removed through a needle in a procedure called arthrocentesis. Not only supports extracting fluid in the diagnosis of this disease, but it can also reduce the pressure in the joint, thereby reducing pain. If the crystals are not found in the translation, gout diagnosis cannot be made with certainty. Occasionally, the crystals cannot be seen for the first time, but can be seen if additional liquid is removed in a subsequent attack.
- Blood tests. A high level of uric acid in your blood does not necessarily mean you have gout, just an ordinary degree does not mean you do not have it.
Blood test results can be confusing, though. Some people have high uric acid levels, but never having gout.
- X-ray images. Part X-rays may be helpful to exclude other causes of arthritis.
- Supersonic. Musculoskeletal ultrasound can detect urate crystals in the joints or in a tophus.
- Dual energy CT scan. This type of image can detect the presence of urate crystals in the joints, even if it is not deep inflammation. Because of high cost and not popular, this test is not used frequently.
- Other symptoms: patients taking drugs; the diet of the patient; How quickly and strongly the attacks of this disease came on; level of pain, the length of the attack and the affected joints.
- Medicines to treat gout
Drugs used to treat acute attacks and prevent attacks in the future include:
- Nonsteroidal anti-inflammatory drugs (NSAIDs). NSAIDs include optional over-the-counter, such as ibuprofen and naproxen sodium or prescription NSAID indomethacin-powerful (Indocin) or celecoxib (Celebrex). NSAIDs carry the risk of stomach pain, bleeding and ulcers. However, do not take aspirin, which should never be used to reduce pain in a gout attack. Be safe with medicines by consulting your doctor and reading carefully the instructions.
- Colchicine helps reduce the pain and swelling of an acute attack. Your doctor may recommend colchicine (Colcrys, Mitigare), an effective analgesic pain relieving gout. However, doctor usually prescribe a low quantity because of its side effects.
- Corticosteroids: such as prednisone is used in pill form to control gout inflammation and pain. It is also the alternative of NSAIDs or colchicine.
- Medicines to prevent complications of gouty arthritis
This method is recommended by doctors if you encounter multiple gout attacks each year, or if your gout attacks are less common but especially pain:
- The drugs block production of uric acid. Function of limit the amount of uric to reduce the risk of gout.
- Drugs can improve the removal of uric acid: reduce your uric acid levels and reduce the risk of gout, instead make the level of uric acid in the urine increase. It may cause rash, stomach pain and kidney stones.
- Gouty arthritis – Surgery
If the symptoms of gout has happened and do not need treatment for more than 10 years, uric acid crystals may have been built-up in the joints to form gritty, chalk marks called tophi. If tophi are causing the infection, pain, pressure, and deformed joints, your doctor may be able to treat them with drugs. But the last choice of surgery must be recommended to remove them if other treatments make no use.
- Lifestyle and home remedies
There is no doubt that the most helpful treatment for gout patients is changing your lifestyle and learn to do some home remedies to make the life better and easier.
- Limiting alcoholic drinks, especially beer and soft drinks with fruit sugar (fructose). The uric acid in your body may increase because alcohol prevent them from release by the kidneys. Beer, is rich in purines, seems worse than some other beverages that contain alcohol. Instead, drink a lot of alcoholic beverages, especially water.
- Restrict purines foods such as red meat, organ meats and seafood.
- Regular exercise and weight loss. Keep your body at a healthy weight reduces the risk of gout.
- Affected joint position until the attack soothe and for 24 hours after the attacks.
- Improving joint pain. Use ice to reduce swelling.
- Continue your medications to gout. Relieve arthritis by using nonsteroidal anti-inflammatory drug (NSAID). Be careful with medicines by consulting your doctor. Some drugs will not help reduce the acute pain. Read and follow all label instructions.
- Follow a program of moderate exercise.
- Restricting meat and seafood. A diet high in meat and seafood (high-purine foods-) can increase uric acid levels.